McDonald’s long shied away from talking about how it makes the food on its menu and what is in that food. The company is now being open and loud about it.
At an event Monday at its headquarters here, McDonald’s announced several changes to its ingredients, including eliminating artificial preservatives from some breakfast foods and Chicken McNuggets, its most popular food item, and removing high-fructose corn syrup from its buns.
Such changes, together with its decision in 2015 to buy only chicken raised without antibiotics used to treat humans, affect almost half of the food on McDonald’s menu, the company said.
I’m going to talk about something completely different than the usual obesity, insulin and type 2 diabetes stuff – antibiotics. This is another area where current medical teaching is completely logic-free. In many ways it reminds me of the entire ‘Type 2 diabetic patients have too much insulin. So, let’s give them more insulin and see if it helps’ argument. Logically it makes no sense. So, instead the medical establishment adopts a ‘I’m the expert so don’t bother trying to talk sense into me. Just do what I say’ attitude.
Antibiotic treatment regimens are largely the same. Suppose you go to your physician for a bacterial infection. Viruses, like most common colds, are not affected by antibiotics, so therefore should not be prescribed. However, because many bacterial infections have the same symptoms, antibiotics are often prescribed ‘just in case’. This leads to antibiotic overuse.
Obesity and diabetes are so common these days, doctors often refer to them as diabesity. Her’e’s a small Canadian study in the SAMJ that posits a whole new paradigm in research to treat diabesity. It’s a path filled with life-saving promise of ‘a ‘cure’ for obesity and diabetes. In Part 1 of a two-part series, Foodmed.net looks at why this study by Canadian and ex-pat South African doctors just may live up to the authors’ hopes and dreams of a real breakthrough. We also look at why establishment doctors and dietitians are resisting the study’s message with all their might.
All this is based on the opinions of the infamous researcher Sof Andrikopoulos. Yes, the same one who “proved” Paleo diets cause diabetes and obesity in humans, by feeding mice sugar and canola oil.
So how can you prove that humans are harmed by Paleo, by feeding mice something that is not Paleo? Beats me. You’ll have to ask Andrikopoulos, because it makes zero sense to me.
Anyway, now Andrikopoulos is again claiming Paleo is bad, as it does not have enough long-term evidence for a positive effect in diabetes type 2. And obviously more scientific backing would be great. But conventional treatment does not just lack the same evidence, what evidence there is shows it works really badly, and the track record is abysmal.
We know how important gut health is for overall health. We understand that it improves digestion, that our pursuit of extreme sterility has compromised our immune systems, and that the gut biome is etiologically involved in the pathogenesis of various health and disease states. We’re even familiar with the more esoteric functions of gut bacteria, like converting antinutrients into biovailable nutrients, synthesizing sex hormones and neurotransmitters, and mitigating the allergenicity of gluten. But what about gaining and losing body fat, the real reason most people get interested in diet in the first place—are the bacteria in your gut responsible for the fat on it?
Crohn’s disease is regarded as having no curative treatment. Previous reports on dietary therapy of Crohn’s disease indicate no major success. Case Report: Here we report a severe case of Crohn’s disease where we successfully applied the paleolithic ketogenic diet. Dietary therapy resulted in resolution of symptoms, normalized laboratory parameters as well as gradual normalization of bowel inflammation as evidenced by imaging data and normalization of intestinal permeability as shown by the polyethylene glycol (PEG 400) challenge test. The patient was able to discontinue medication within two weeks. Currently he is on the diet for 15 months and is free of symptoms as well as side effects. Conclusion: We conclude that the paleolithic ketogenic diet was feasible, effective and safe in the present case.
The UKPDS (United Kingdom Prospective Diabetes Study) was a huge study undertaken in the UK to see if intensive blood glucose lowering in T2D would prevent end organ damage over long run. The DCCT study mentioned previously had already established the paradigm of tight blood sugar control in Type 1, but whether this held true for type 2 remained to be seen.
3867 newly diagnosed T2D patients who failed a 3 month lifestyle therapy trial were enrolled into an intensive group with sulfonlyureas or insulin versus conventional control (UKPDS 33). The intensive group would target a fasting glucose of less than 6.0 mmol/L. In the conventional group, drugs were only added if FBG exceeded 15. If high blood sugars was the primary cause of disease, then this intensive group should do better. We can move the sugar from the blood into the body with drugs, but the price to be paid is excessively high insulin levels. Remember that these T2D patients had a baseline level of insulin that was already high. We would raise them even further in order to lower blood sugars.